How fast does sudden cardiac arrest cause unconsciousness? In just seconds.
Here’s a video of Salamanca soccer player Miguel Garcia’s episode. At the start of the video, Mr. Garcia can be seen in the background of the image kneeling behind the players in the foreground. Watch carefully as he stands after tying his shoes.
Although it is difficult to see, it appears an automatic external defibrillator arrives in about two minutes, though given the fact his shirt is still on as he’s taken from the field, we note the device is on his gurney as he’s hurried to a nearby ambulance. Reportedly, he survived this sudden cardiac arrest event:
This was NOT a heart attack, but rather a loss of cardiac function caused by a rapid, often disorganized heart rhythm disorder. Compare the relatively long time to resuscitation using an external automatic defibrillator verses the very rapid response afforded to Belgian soccer player Anthony Van Loo, whose internal defibrillator was already installed before he played as primary prevention of sudden death from right ventricular dysplasia.
-WesMusings of a cardiologist and cardiac electrophysiologist.
Imagine jogging, listening to music, and being able to keep track of your heart rate without needing a special watch or chest belt — common forms of attempting to monitor heart rates while jogging. Now, imagine not requiring any extra peripherals at all — just your iPhone and a special set of headphones that can monitor your heart rate.
Swiss technology-transfer company CSEM has created the final prototype for their Pulsear device. It’s a tiny device embedded in a regular earphone and it sends infrared signals through the tissues in your ear to see how fast your heart is beating. A photo diode records the results and sends the information to your phone via the earphone wires. Read more »
*This blog post was originally published at iMedicalApps*
In psychiatry, we’ve had a hard time drawing precise links between brain pathology and psychiatric disorders. We can do it for groups of people: “Disease X” is associated with changes in brain structure of “Brain Area Y” or metabolic changes in “Brain Area Z.” But it’s groups, not individuals, and it’s an association, not a cause-and-effect, or a definite. We still can’t use this information for diagnosis, and there are still patients with any given psychiatric diagnoses who will have brains where “Area Y” is the same size as those without the disorder. We’re learning.
From what I read in this New York Times article, Owen Thomas was a bright, talented young man with no history of psychiatric disorder and no history of known concussion. In April, he committed suicide — a tragedy beyond words.
Sometime people commit suicide and everyone is left to wonder: There was no depression, no obvious precipitant, no note left behind, and every one is left to wonder why. The guilt toll on the survivors is enormous, as is the grief for their families and communities. In this case, according to the Philadelphia Inquirer, the young man was apparently struggling with the stress of difficult school work and concerns about his team and employment.
Owen’s family donated his brain to Boston University’s Center for the Study of Traumatic Encephalopathy. They discovered that Owen’s brain showed damage similar to that seen in older NFL players — he had a condition called chronic traumatic encephalopathy. Read more »
*This blog post was originally published at Shrink Rap*
The guy next to me on the bike yesterday morning was working like Lance Armstrong in training: He had laid towels on the floor to absorb the impressive perspiration he was generating.
He shouted over to me: “I’m hitting it hard to cleanse out the toxins from last night. Too much Captain Morgan and buffalo wings, ya know?”
“Really,” I said.
“Actually, I’m a dermatologist, and sweat does not contain any toxins,” I said to myself so that he could not hear. (Gym decorum dictates men do not correct men in the middle of a workout — especially if prefaced by “Actually, I’m a dermatologist.”) I left him to his aerobics and wrote this post in my head while I finished mine. Read more »
It’s the dog days of what seems to have been an unusually hot summer (though DrRich does not know whether it has been sufficiently warm to affect the global cooling trend we’ve been in for the past decade), and as is all too common at this time of year, we are seeing extraordinarily heartbreaking stories (like this one) about healthy, robust young athletes dying suddenly on the practice fields.
Most of these tragic sudden deaths are due to a heart condition called hypertrophic cardiomyopathy. Hypertrophic cardiomyopathy often does not produce any symptoms prior to causing sudden death. But it can be easily diagnosed, before exercise-induced sudden death occurs, by screening young athletes with electocardiograms (ECGs) and echocardiography.
A couple of summers ago, the New York Times wrote about such an athletic screening program at the University of Tennessee. Based on the U of T’s results, “cardiologists and other heart experts say that the screenings could help save the lives of the 125 American athletes younger than 35 who die each year of sudden cardiac death.” Read more »
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In psychiatry, we’ve had a hard time drawing precise links between brain pathology and psychiatric disorders. We can do it for groups of people: “Disease X” is associated with changes in brain structure of “Brain Area Y” or metabolic changes in “Brain Area Z.” But it’s groups, not individuals, and it’s an association, not a cause-and-effect, or a definite. We still can’t use this information for diagnosis, and there are still patients with any given psychiatric diagnoses who will have brains where “Area Y” is the same size as those without the disorder. We’re learning.
Can you sweat out toxins?
