It looks like New York City is leading the way for public health safety by introducing a public smoking ban in all public parks, malls, plazas, beaches and playgrounds or risk a $50 fine:
Research showed, he said, that someone seated within three feet of a smoker — even in the open air — was exposed to roughly the same levels of secondhand smoke as someone sitting indoors in the same situation.
What took so long? Go, New York. I hope you succeed. Next up: Charging parents who smoke in their homes occupied by minors with child negligence.
(Guest post submitted by MD Anderson Cancer Center)
Aisles in grocery stores and pharmacies are stacked with vitamins, minerals, herbs or other plants that you take in pill, capsule, tablet or liquid form. And, many of us buy these supplements and take them regularly, hoping to lower our chances of getting cancer and other diseases.
But do supplements really work wonders? Should you take them to help prevent cancer? Our experts say beware.
“Don’t be fooled by the label on the bottle,” says Sally Scroggs, health education manager at MD Anderson’s Cancer Prevention Center. “Researchers are still unsure about whether or not supplements actually prevent cancer.” Some studies have suggested that supplements may actually increase cancer risk by tilting the balance of nutrients in the body. Read more »
*This blog post was originally published at Health in 30*
In psychiatry, we’ve had a hard time drawing precise links between brain pathology and psychiatric disorders. We can do it for groups of people: “Disease X” is associated with changes in brain structure of “Brain Area Y” or metabolic changes in “Brain Area Z.” But it’s groups, not individuals, and it’s an association, not a cause-and-effect, or a definite. We still can’t use this information for diagnosis, and there are still patients with any given psychiatric diagnoses who will have brains where “Area Y” is the same size as those without the disorder. We’re learning.
From what I read in this New York Times article, Owen Thomas was a bright, talented young man with no history of psychiatric disorder and no history of known concussion. In April, he committed suicide — a tragedy beyond words.
Sometime people commit suicide and everyone is left to wonder: There was no depression, no obvious precipitant, no note left behind, and every one is left to wonder why. The guilt toll on the survivors is enormous, as is the grief for their families and communities. In this case, according to the Philadelphia Inquirer, the young man was apparently struggling with the stress of difficult school work and concerns about his team and employment.
Owen’s family donated his brain to Boston University’s Center for the Study of Traumatic Encephalopathy. They discovered that Owen’s brain showed damage similar to that seen in older NFL players — he had a condition called chronic traumatic encephalopathy. Read more »
*This blog post was originally published at Shrink Rap*
The Washington Postasks whether “old age” should be reconsidered as a legitimate cause of death for the elderly. Because more people are dying at very advanced ages with multiple system failure, it’s often harder for physicians to pinpoint the specific underlying cause, but using “old age” as a catch-all term could make mortality data less meaningful, the article said.
An upcoming revision of the International Classification of Diseases might provide some guidance: “Each revision of the ICD is the right moment to reconsider this question,” the co-head of the ICD’s mortality statistics committee told the Post. (Washington Post)
*This blog post was originally published at ACP Internist*
Last month I reported about the Social Media Code of Conduct released by Roche and I also shared my opinion on the issue. Now Sabine Kostevc, Head of Corporate Internet and Social Media at F. Hoffmann-La Roche AG, gave an interview to Silja Chouquet:
*This blog post was originally published at ScienceRoll*
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In psychiatry, we’ve had a hard time drawing precise links between brain pathology and psychiatric disorders. We can do it for groups of people: “Disease X” is associated with changes in brain structure of “Brain Area Y” or metabolic changes in “Brain Area Z.” But it’s groups, not individuals, and it’s an association, not a cause-and-effect, or a definite. We still can’t use this information for diagnosis, and there are still patients with any given psychiatric diagnoses who will have brains where “Area Y” is the same size as those without the disorder. We’re learning.
